1999; 15: 2376-2383.
[2] Takahashi K, Mulliken JB, et al. Cellular markers that distinguish the phase of hemangioma during infancy and childhood. Clin Invist. 1994; 93: 2357. [3] 王康敏, 孙亚丽, 等. VEGF、bFGF、C-myc在婴幼儿皮肤血管瘤中的作用及相关性. 中国皮肤性病学杂志. 2005; 19: 9-11.
[4] Chang J, Most D. Proliferative hemangiomasl: analysis of cytokine gene expression and angiogenesis. Plast Reconstr Surg. 1999; 103: 1-9.
[5] Helen E, Turner, et al. Angiogenesis in Endocrine Tumors. The Endocrine Society. 2008; 24: 600-632.
[6] Gately S, The contributions of cyclooxygenase-2 to tumor angiogenesis. Cancer Metastasis Rev. 2000; 19: 19-27.
[7] Masatoshi J, Damian M, et al. Suppressed NFAT-dependent VEGFR1 expression and constitutive VEGFR2 signaling in infantile hemangioma. Nature Medicine. 2008; 14: 1236-1246.
[8] 张莉, 张成书. 基因芯片技术筛选血管瘤发生相关基因. 蚌埠医学院学报. 2008; 33: 668-671.
[9] McLaughlin B, Hartnett KA, et al. Caspase-3 activation is essential for neuroprotection in preconditioning. PNAS. 2003; 100: 715-720.
[10] Kishi H, Igawa M, et al. Expression of surviving gene in prostate cancer: correlation with clinicopathological characteristics,proliferative activity and apoptosis. Jurol. 2004; 171: 1855.
[11] Giulia T, Laura S, et al. Posttranscriptional stimulation of endothelial cell matrixmetalloproteinase-2 and-1 by endothelioma cells. Experimental cell research. 2000; 258: 384-394.
[12] 黄辉, 罗莉. 雌二醇在婴幼儿血管瘤及血管畸形中的表达及意义. 现代医药卫生. 2007; 23: 1132-1134.